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Do Black People Shed Dark Skin Cells

Human pare color

Dark pare is a blazon of human skin color that is rich in melanin pigments, specially eumelanin.[ane] [2] [3] People with very dark pare are often referred to as "black people",[4] although this usage tin be ambiguous in some countries where information technology is likewise used to specifically refer to dissimilar ethnic groups or populations.[5] [6] [7] [8]

The evolution of nighttime peel is believed to accept begun around 1.2 million years agone,[9] in lite-skinned early hominid species subsequently they moved from the equatorial rainforest to the sunny savannas. In the heat of the savannas, meliorate cooling mechanisms were required, which were achieved through the loss of torso hair and development of more than efficient perspiration. The loss of body hair led to the development of nighttime pare pigmentation, which acted every bit a mechanism of natural selection against folate depletion, and to a bottom extent, Deoxyribonucleic acid damage. The primary factor contributing to the evolution of dark skin pigmentation was the breakdown of folate in reaction to ultraviolet radiation; the relationship betwixt folate breakdown induced by ultraviolet radiation and reduced fitness as a failure of normal embryogenesis and spermatogenesis led to the selection of night skin pigmentation. By the time modernistic Homo sapiens evolved, all humans were dark-skinned.[3] [ten] [11] [12] [13] [14] [15]

Humans with dark skin pigmentation have skin naturally rich in melanin (especially eumelanin), and accept more melanosomes which provide superior protection against the deleterious effects of ultraviolet radiation. This helps the trunk to retain its folate reserves and protects against impairment to Dna.[iii] [16]

Dark-skinned people who alive in loftier latitudes with mild sunlight are at an increased chance—particularly in the wintertime—of vitamin D deficiency. As a consequence of vitamin D deficiency, they are at a higher chance of developing rickets, numerous types of cancers, and peradventure cardiovascular disease and depression allowed system action.[3] [17] However, some recent studies have questioned if the thresholds indicating vitamin D deficiency in light-skinned individuals are relevant for dark-skinned individuals, as they establish that, on average, dark-skinned individuals have higher bone density and lower hazard of fractures than lighter-skinned individuals with the same levels of vitamin D. This is possibly attributed to lower presence of vitamin D bounden agents (and thus its higher bioavailability) in dark-skinned individuals.[18] [19]

The global distribution of generally dark-skinned populations is strongly correlated with the loftier ultraviolet radiation levels of the regions inhabited past them. These populations, with the exception of indigenous Tasmanians almost exclusively live nigh the equator, in tropical areas with intense sunlight: Australia, Melanesia, New Guinea, South Asia, Southeast Asia, West Asia, and Africa. Studies into these populations indicates dark peel is a retention of the pre-existing high UVR-adjusted country of mod humans before the out of Africa migration and not a later evolutionary accommodation.[20] [21] Due to mass migration and increased mobility of people between geographical regions in the recent by, night-skinned populations today are establish all over the world.[3] [22] [23]

Evolution [edit]

Due to natural choice, people who lived in areas of intense sunlight adult dark pare colouration to protect against ultraviolet (UV) light, mainly to protect their torso from folate depletion. Evolutionary pigmentation of the skin was caused past ultraviolet radiation of the sun. As hominids gradually lost their fur between 1.2 and 4 million years ago, to allow for better cooling through sweating, their naked and lightly-pigmented skin was exposed to sunlight. In the tropics, natural selection favoured dark-skinned human populations equally high levels of skin pigmentation protected confronting the harmful furnishings of sunlight. Indigenous populations' pare reflectance (the amount of sunlight the pare reflects) and the actual UV radiations in a particular geographic area is highly correlated, which supports this idea. Genetic evidence likewise supports this notion, demonstrating that around 1.2 million years ago there was a strong evolutionary force per unit area which acted on the development of dark skin pigmentation in early members of the genus Homo.[24] The result of sunlight on folic acid levels has been crucial in the evolution of nighttime peel.[three] [25]

The earliest primate ancestors of mod humans most likely had light skin, like our closest mod relative—the chimpanzee.[26] About 7 one thousand thousand years ago homo and chimpanzee lineages diverged, and betwixt 4.5 and 2 million years ago early on humans moved out of rainforests to the savannas of East Africa.[22] [27] They non only had to cope with more than intense sunlight just had to develop a amend cooling organization. It was harder to go nutrient in the hot savannas and every bit mammalian brains are decumbent to overheating—5 or vi °C rise in temperature tin lead to heatstroke—at that place was a need for the evolution of improve heat regulation. The solution was sweating and loss of body hair.[22]

Sweating dissipated heat through evaporation. Early humans, similar chimpanzees now, had few sweat glands, and most of them were located in the palms of the hand and the soles of the anxiety. At times, individuals with more sweat glands were built-in. These humans could search for food and hunt for longer periods before being forced back to the shades. The more than they could forage, the more and healthier offspring they could produce, and the college the chance they had to laissez passer on their genes for abundant sweat glands. With less pilus, sweat could evaporate more than easily and cool the bodies of humans faster. A few million years of evolution later, early humans had sparse body hair and more than than 2 million sweat glands in their torso.[22] [28] [29]

Hairless skin, however, is particularly vulnerable to exist damaged by ultraviolet light and this proved to be a problem for humans living in areas of intense UV radiation, and the evolutionary result was the evolution of nighttime-coloured pare equally a protection. Scientists have long assumed that humans evolved melanin in lodge to blot or scatter harmful sun radiation. Some researchers assumed that melanin protects confronting peel cancer. While loftier UV radiation can cause peel cancer, the evolution of cancer ordinarily occurs after child-bearing age. As natural selection favours individuals with traits of reproductive success, pare cancer had trivial effect on the evolution of dark peel. Previous hypotheses suggested that sunburned nipples impeded breastfeeding, just a slight tan is enough to protect mothers against this issue.[22] [30] [31] [32]

A 1978 report examined the consequence of sunlight on folate—a vitamin B circuitous—levels.[ citation needed ] The study found that fifty-fifty brusk periods of intense sunlight are able to halve folate levels if someone has light skin. Low folate levels are correlated with neural tube defects, such as anencephaly and spina bifida. UV rays can strip away folate, which is important to the development of salubrious foetuses. In these abnormalities children are born with an incomplete encephalon or spinal cord. Nina Jablonski, a professor of anthropology and expert on evolution of homo peel coloration,[33] found several cases in which mothers' visits to tanning studios were connected to neural tube defects in early on pregnancy. She also institute that folate was crucial to sperm development; some male contraception drugs are based on folate inhibition. It has been plant that folate may have been the driving strength behind the evolution of nighttime skin.[three] [20]

As humans dispersed from equatorial Africa to low UVR areas and higher latitudes sometime betwixt 120,000 and 65,000 years agone, dark skin posed a disadvantage.[34] [35] Populations with light pare pigmentation evolved in climates of footling sunlight. Low-cal skin pigmentation protects against vitamin D deficiency. It is known that nighttime-skinned people who have moved to climates of limited sunlight can develop vitamin D-related weather condition such every bit rickets, and dissimilar forms of cancer.[3] [36]

Earlier hypotheses [edit]

The main other hypotheses that take been put forward through history to explicate the development of nighttime skin coloration relate to increased bloodshed due to pare cancers, enhanced fitness every bit a result of protection against sunburns, and increasing benefits due to antibacterial properties of eumelanin.[3]

Darkly pigmented, eumelanin-rich pare protects against Deoxyribonucleic acid damage caused by the sunlight.[37] This is associated with lower skin cancer rates among dark-skinned populations.[38] [39] [40] [41] [42] The presence of pheomelanin in low-cal skin increases the oxidative stress in melanocytes, and this combined with the express ability of pheomelanin to absorb UVR contributes to higher peel cancer rates among light-skinned individuals.[43] The damaging effect of UVR on DNA construction and the entailing elevated skin cancer risk is widely recognized.[24] [44] [45] [46] [47] However, these cancer types normally bear upon people at the stop or after their reproductive career and could have not been the evolutionary reason behind the evolution of dark skin pigmentation.[24] [31] Of all the major skin cancer types, only malignant melanoma have a major effect in a person's reproductive historic period. The mortality rates of melanoma has been very low (less than 5 per 100,000) earlier the mid-20th century. It has been argued that the low melanoma mortality rates during reproductive age cannot be the principal reason backside the evolution of nighttime pare pigmentation.[32]

Studies take institute that even serious sunburns could non bear on sweat gland part and thermoregulation. In that location are no data or studies that support that sunburn can cause damage and then serious information technology can bear on reproductive success.[3]

Another group of hypotheses contended that night peel pigmentation developed as antibacterial protection against tropical infectious diseases and parasites. Although it is true that eumelanin has antibacterial properties, its importance is secondary to 'physical adsorption' (physisorption) to protect against UVR-induced harm. This hypothesis is non consistent with the evidence that most of the hominid evolution took place in savanna environments and not in tropical rainforests.[48] Humans living in hot and sunny environments take darker skin than humans who live in wet and cloudy environments.[35] The antimicrobial hypothesis also does not explain why some populations (like the Inuit or Tibetans) who alive far from the torrid zone and are exposed to high UVR accept darker skin pigmentation than their surrounding populations.[3]

Biochemistry and genetics [edit]

Darkly pigmented skin

Nighttime-skinned humans have high amounts of melanin constitute in their skin. Melanin is derivative of the amino acid tyrosine. Eumelanin is the ascendant form of melanin found in homo skin. Eumelanin protects tissues and Deoxyribonucleic acid from the radiations damage of UV light. Melanin is produced in specialized cells chosen melanocytes, which are institute at the everyman level of the epidermis.[49] Melanin is produced within modest membrane-bound packages called melanosomes. People with naturally-occurring dark skin have melanosomes which are clumped, large and full of eumelanin.[50] [51] A 4-fold difference in naturally-occurring dark pare gives seven- to 8-fold protection against Dna damage,[51] but fifty-fifty the darkest skin colour cannot protect confronting all impairment to Dna.[three]

Dark skin offers groovy protection confronting UVR considering of its eumelanin content, the UVR-absorbing capabilities of big melanosomes, and because eumelanin can be mobilized faster and brought to the surface of the pare from the depths of the epidermis.[3] For the same torso region, light- and dark-skinned individuals have similar numbers of melanocytes (in that location is considerable variation between different torso regions), simply paint-containing organelles, chosen melanosomes, are larger and more numerous in dark-skinned individuals. Keratocytes from night skin cocultured with melanocytes give rise to a melanosome distribution pattern feature of dark skin.[52] [53] Melanosomes are non in aggregated state in darkly pigmented skin compared to lightly pigmented pare. Due to the heavily melanised melanosomes in darkly-pigmented skin, it tin absorb more energy from UVR and thus offers better protection against sunburns and by absorption and dispersion UV rays.[24]

Darkly-pigmented skin protects confronting direct and indirect DNA impairment. Photodegration occurs when melanin absorbs photons. Recent enquiry advise that the photoprotective effect of night peel is increased by the fact that melanin can capture complimentary radicals, such as hydrogen peroxide, which are created by the interaction of UVR and layers of the pare.[24] Heavily pigmented melanocytes take greater chapters to carve up after ultraviolet irradiation, which suggests that they receive less impairment to their Dna.[24] Despite this, medium-wave ultraviolet radiations (UVB) damages the allowed system fifty-fifty in darker skinned individuals due to its event on Langerhans cells.[24] The stratum corneum of people with dark or heavily tanned peel is more than condensed and contains more cornified cell layers than in lightly-pigmented humans. These qualities of nighttime skin enhance the barrier protection function of the skin.[24]

Although darkly-pigmented peel absorbs nearly xxx to forty% more sunlight than lightly-pigmented skin, dark skin does not increase the trunk's internal heat intake in conditions of intense solar radiations. Solar radiation heats up the body'due south surface and not the interior. Furthermore, this amount of heat is negligible compared to the heat produced when muscles are actively used during exercise. Regardless of peel colour, humans have excellent capabilities to dissipate rut through sweating.[35] Half of the solar radiations reaching the World'due south surface is in the course of infrared lite and is absorbed similarly regardless of pare coloration.[24]

In people with naturally occurring night skin, the tanning occurs with the dramatic mobilization of melanin upward in the epidermis and continues with the increased product of melanin. This accounts for the fact that night-skinned people get visibly darker afterward i or two weeks of sun exposure, and then lose their colour later on months when they stay out of the sun. Darkly-pigmented people tend to showroom fewer signs of aging in their skin than the lightly-pigmented because their night pare protects them from nearly photoaging.[35]

Skin color is a polygenic trait, which means that several different genes are involved in determining a specific phenotype. Many genes work together in complex, additive, and not-condiment combinations to decide the skin colour of an private. The skin colour variations are normally distributed from calorie-free to dark, as it is usual for polygenic traits.[54] [55]

Data collected from studies on MC1R gene has shown that there is a lack of diversity in dark-skinned African samples in the allele of the gene compared to non-African populations. This is remarkable given that the number of polymorphisms for almost all genes in the human factor pool is greater in African samples than in any other geographic region. So, while the MC1Rf gene does not significantly contribute to variation in skin color around the globe, the allele constitute in high levels in African populations probably protects against UV radiations and was probably of import in the development of dark pare.[56] [57]

Skin colour seems to vary mostly due to variations in a number of genes of big result also as several other genes of small effect (TYR, TYRP1, OCA2, SLC45A2, SLC24A5, MC1R, KITLG and SLC24A4). This does non accept into business relationship the effects of epistasis, which would probably increase the number of related genes.[58] Variations in the SLC24A5 gene account for twenty–25% of the variation between dark- and light-skinned populations of Africa,[59] and appear to take arisen as recently as within the last 10,000 years.[60] The Ala111Thr or rs1426654 polymorphism in the coding region of the SLC24A5 cistron reaches fixation in Europe, and is besides common amidst populations in Northward Africa, the Horn of Africa, West Asia, Central Asia and South asia.[61] [62] [63]

Health implications [edit]

Skin pigmentation is an evolutionary adaptation to various UVR levels effectually the world. As a issue there are many health implications that are the product of population movements of humans of certain skin pigmentation to new environments with different levels of UVR.[iii] Modern humans are often ignorant of their evolutionary history at their peril.[3] Cultural practices that increase problems of conditions among dark-skinned populations are traditional clothing and vitamin D-poor diet.[64]

Advantages in high sunlight [edit]

Dark-pigmented people living in high sunlight environments are at an reward due to the high amounts of melanin produced in their skin. The dark pigmentation protects from Deoxyribonucleic acid damage and absorbs the right amounts of UV radiation needed by the body, as well as protects against folate depletion. Folate is a water-soluble vitamin B complex which naturally occurs in green, leafy vegetables, whole grains, and citrus fruits. Women need folate to maintain good for you eggs, for proper implantation of eggs, and for the normal development of placenta afterward fertilization. Folate is needed for normal sperm production in men. Furthermore, folate is essential for fetal growth, organ development, and neural tube development. Folate breaks down in high intensity UVR.[35] Dark-skinned women suffer the lowest level of neural tube defects.[35] [65] Folate plays an important office in Deoxyribonucleic acid production and factor expression. Information technology is essential for maintaining proper levels of amino acids which brand up proteins. Folate is used in the formation of myelin, the sheath that covers nerve cells and makes information technology possible to send electrical signals quickly. Folate also plays an important role in the development of many neurotransmitters, east.g. serotonin which regulates appetite, sleep, and mood. Serum folate is broken downward by UV radiation or alcohol consumption.[35] Because the skin is protected by the melanin, dark-pigmented people have a lower chance of developing peel cancer and conditions related to folate deficiency, such as neural tube defects.[3]

Disadvantages in low sunlight [edit]

Rickets is a condition associated with night skin.

Dark-skinned people living in low sunlight environments have been recorded to be very susceptible to vitamin D deficiency due to reduced vitamin D synthesis. A dark-skinned person requires about six times as much UVB than lightly-pigmented persons. This is not a problem nigh the equator; even so, it can be a problem at college latitudes.[35] For humans with dark peel in climates of low UVR, it can take about two hours to produce the same corporeality of vitamin D as humans with lite skin produce in 15 minutes. Dark-skinned people having a high body-mass index and not taking vitamin D supplements were associated with vitamin D deficiency.[66] [67] Vitamin D plays an important office in regulating the human immune system and chronic deficiencies in vitamin D can make humans susceptible to specific types of cancers and many kinds of infectious diseases.[35] [68] [69] Vitamin D deficiency increases the gamble of developing tuberculosis five-fold and as well contributes to the development of breast, prostate, and colorectal cancer.[lxx]

The almost prevalent affliction to follow vitamin D deficiency is rickets, the softening of bones in children potentially leading to fractures and deformity.[ citation needed ] Rickets is acquired by reduced vitamin D synthesis that causes an absenteeism of vitamin D, which so causes the dietary calcium to not exist properly absorbed. This disease in the by was ordinarily establish among dark-skinned Americans of the southern role of the Usa who migrated north into depression sunlight environments. The popularity of sugary drinks and decreased time spent outside accept contributed to significant rising of developing rickets. Deformities of the female pelvis related to severe rickets impair normal childbirth, which leads to higher mortality of the infant, mother, or both.

Vitamin D deficiency is most common in regions with depression sunlight, especially in the wintertime.[71] Chronic deficiencies in vitamin D may also be linked with breast, prostate, colon, ovarian, and possibly other types of cancers.[22] [72] [73] [74] The relationship between cardiovascular disease and vitamin D deficiency too suggest a link betwixt health of cardiac and shine musculus.[75] [76] Low vitamin D levels have besides been linked to impaired immune system and brain functions.[iii] [77] [78] In addition, recent studies take linked vitamin D deficiency to autoimmune diseases, hypertension, multiple sclerosis, diabetes and incidence of retentiveness loss.

Outside the torrid zone UVR has to penetrate through a thicker layer of atmosphere, which results in well-nigh of the intermediate wavelength UVB reflected or destroyed en route; because of this there is less potential for vitamin D biosynthesis in regions far from the equator. Higher amount of vitamin D intake for dark-skinned people living in regions with depression levels of sunlight are advised past doctors to follow a vitamin D-rich diet or have vitamin D supplements,[22] [79] [80] [81] [82] [83] although at that place is recent evidence that dark-skinned individuals are able to process vitamin D more efficiently than lighter-skinned individuals so may have a lower threshold of sufficiency.[19]

Geographic distribution [edit]

At that place is a correlation between the geographic distribution of UV radiations (UVR) and the distribution of skin pigmentation around the world. Areas that have college amounts of UVR have darker-skinned populations, generally located nearer the equator. Areas that are further away from the equator and mostly closer to the poles have a lower concentration of UVR and incorporate lighter-skinned populations. This is the result of man evolution which contributed to variable melanin content in the skin to adapt to sure environments. A larger percentage of dark-skinned people are found in the Southern Hemisphere because latitudinal state mass distribution is disproportionate.[24] The present distribution of skin colour variation does not completely reflect the correlation of intense UVR and dark skin pigmentation due to mass migration and movement of peoples across continents in the recent past.[24] Dark-skinned populations inhabiting Africa, Australia, Melanesia, Papua New Guinea and South Asia all live in some of the areas with the highest UV radiations in the earth, and have evolved very dark pare pigmentations as protection from the dominicus's harmful rays.[22] [24] Evolution has restricted humans with darker skin in tropical latitudes, especially in non-forested regions, where ultraviolet radiation from the sun is usually the near intense. Different dark-skinned populations are not necessarily closely related genetically.[84] Before the modern mass migration, it has been argued that the majority of dark-pigmented people lived within 20° of the equator.[85]

Natives of Buka and Bougainville at the northern Solomon Islands in Melanesia and the Chopi people of Mozambique in the southeast declension of Africa have darker peel than other surrounding populations. (The native people of Bougainville, Papua New Guinea, have some of the darkest skin pigmentation in the world.) Although these people are widely separated they share similar physical environments. In both regions, they feel very loftier UVR exposure from cloudless skies about the equator which is reflected from water or sand. Water reflects, depending on color, about 10 to 30% of UVR that falls on it.[35] [86] People in these populations spend long hours fishing on the sea. Considering it is impractical to wear extensive vesture in a watery environment, civilisation and technology does picayune to buffer UVR exposure. The skin takes a very large corporeality of ultraviolet radiation. These populations are probably about or at the maximum darkness that human skin tin achieve.[35]

More recent research has found that human populations over the past l,000 years have changed from nighttime-skinned to light-skinned and vice versa. Simply 100–200 generations ago, the ancestors of most people living today likely likewise resided in a different place and had a different skin colour. According to Nina Jablonski, darkly-pigmented modern populations in South India and Sri Lanka are an case of this, having re-darkened after their ancestors migrated downwardly from areas much farther north. Scientists originally believed that such shifts in pigmentation occurred relatively slowly. However, researchers accept since observed that changes in skin coloration tin happen in equally piddling as 100 generations (~2,500 years), with no intermarriage required. The speed of change is also affected by wear, which tends to slow information technology down.[87]

Australia [edit]

The Aborigines of Australia, every bit with all humans, are descendants of African migrants, and their ancestors may accept been amongst the beginning major groups to exit Africa around 50,000 years ago. Despite early migrations, genetic evidence has pointed out that the indigenous peoples of Australia are genetically very dissimilar to the nighttime-skinned populations of Africa and that they are more closely related to Eurasian populations.[88]

The term black initially has been applied equally a reference to the skin pigmentation of the aborigines of Australia; today it has been embraced past aboriginal activists every bit a term for shared civilisation and identity, regardless of pare colour.[89] [xc]

Melanesia [edit]

Melanesia, a subregion of Oceania, whose name means "black islands", have several islands that are inhabited by people with dark pare pigmentation. The islands of Melanesia are located immediately northward and northeast of Australia too as east coast of Papua New Guinea.[91] The western stop of Melanesia from New Guinea through the Solomon Islands were commencement colonized by humans most 40,000 to 29,000 years ago.[92] [93]

In the world, blond hair is exceptionally rare outside Europe and West Asia, especially among dark-skinned populations. However, Melanesians are i of the dark-skinned human populations known to have naturally-occurring blond hair.[94] [95]

New Guinea [edit]

The indigenous Papuan people of New Guinea have dark skin pigmentation and have inhabited the island for at least forty,000 years. Due to their similar phenotype and the location of New Guinea being in the migration route taken past Indigenous Australians, it was mostly believed that Papuans and Aboriginal Australians shared a common origin. However, a 1999 study failed to find clear indications of a single shared genetic origin betwixt the two populations, suggesting multiple waves of migration into Sahul with distinct ancestries.[96]

Sub-Saharan Africa [edit]

Sub-Saharan Africa is the region in Africa situated due south of the Sahara where a large number of dark-skinned populations live.[97] [98] Night-skinned groups on the continent have the same receptor protein as Homo ergaster and Homo erectus had.[99] According to scientific studies, populations in Africa as well have the highest skin color diversity.[100] High levels of peel color variation exists between different populations in Sub-Saharan Africa. These differences depend in part on general distance from the equator, illustrating the circuitous interactions of evolutionary forces which accept contributed to the geographic distribution of skin color at any point of time.[35]

Due to frequently differing beginnings amongst dark-skinned populations, the presence of dark skin in general is not a reliable genetic marker, including among groups in Africa. For example, Wilson et al. (2001) establish that almost of their Ethiopian samples showed closer genetic affinities with lighter-skinned Armenians than with darker-skinned Bantu populations.[101] Mohamoud (2006) too observed that their Somali samples were genetically more than similar to Arab populations than to other African populations.[102]

Southern asia [edit]

Fisherman from Chennai, Tamil Nadu in southern Bharat.

South Asia has some of the greatest skin color diversity outside of Africa. Peel color amongst Southward Indians is on boilerplate darker than Due north Indians. This is mainly because of the weather condition weather condition in South asia—college UV indices are in the south.[103] Several genetic surveys of Southward Asian populations in different regions have establish a weak negative correlation between social status and skin darkness, represented by the melanin alphabetize. A study of caste populations in the Gangetic Plain found an association betwixt the proportion of nighttime peel and ranking in the caste bureaucracy. Dalits had, on boilerplate, the darkest pare.[104] A pan-Republic of india study of Telugu and N Indian castes plant a similar correlation between pare color and caste association, linked to the absence of the rs1426654-A variant of the SLC245-A gene, just are also linked to mutations overriding these variants.[105]

Americas [edit]

A dark-skinned Wayuu couple from Colombia. Many other Indigenous peoples of tropical or subtropical areas of the Americas have dark skin.

Relatively dark skin remains among the Inuit and other Arctic populations. A combination of poly peptide-heavy diets and summer snowfall reflection have been speculated as favouring the retention of pigmented skin.[3] [100] [25]

Earliest European colonial descriptions of North American populations include terms such as "brown", "tawny" or "olive", though some populations were likewise described equally "light-skinned".[106] Most Due north American indigenous populations rank similar to African and Oceanian populations in regards to the presence of the allele Ala111.[107]

Native South Americans and Mesoamericans are also typically considered dark-skinned, ranking similarly to African and Oceanian populations in regards to Ala111 presence.[107] High ultraviolet radiations levels occur throughout the Andes region of Peru, Republic of bolivia, Republic of chile, and Argentina.[108]

Genetic tests show significant Australian influence,[109] theorizing that Amazonian Indians and Australians both diverged from a common ancestor. Scientists tested the ancient and present-day Genome-wide assay of 49 Central and South Americans up to 11,000 years old from Belize, Brazil, Peru, and the Southern Cone (Chile and Argentina).[110]

Culture [edit]

The preference or disfavour for darker peel has varied depending on geographical area and time. Today, darker skin is viewed as stylish and equally a sign of well-being in some societies. This resulted in the development of tanning industry in several countries. However, in some countries, nighttime skin is not seen as highly desirable or indicative of higher grade, especially among women.[24]

See also [edit]

  • Albinism
  • Black people
  • Dark-brown (racial nomenclature)
  • Olive pare
  • Light skin

References [edit]

  1. ^ dark-skinned Princeton University "naturally having skin of a night color"
  2. ^ "Dark-skinned". thefreedictionary.com. Retrieved 24 Jan 2017. a person or race having pare of a dark color
  3. ^ a b c d e f g h i j g l m n o p q r Muehlenbein, Michael (2010). Human Evolutionary Biology. Cambridge University Press. pp. 192–213.
  4. ^ Oxford Dictionaries. April 2010. Oxford University Press. "belonging to or denoting any homo group having dark-coloured skin" "black" (accessed 6 August 2012).
  5. ^ Dictionary.com: black three.a "a member of any of diverse night-skinned peoples" 21.a"specifically the dark-skinned peoples of Africa, Oceania, or Australia."
  6. ^ "Global Census". American Anthropological Association. Archived from the original on 14 September 2018. Retrieved ten December 2012.
  7. ^ Oxford Dictionaries. Apr 2010. Oxford University Press. "particularly of African or Australian Aboriginal ancestry" "black" (accessed six Baronial 2012).
  8. ^ James, Mackers (8 Nov 1828). "Proclamation". Classified Advertising. Trove. Retrieved 10 December 2012.
  9. ^ Nina, Jablonski (2004). "The evolution of human skin and peel color". Annual Review of Anthropology. 33: 585–623. doi:ten.1146/annurev.anthro.33.070203.143955. S2CID 53481281. genetic show [demonstrate] that stiff levels of natural pick acted about 1.2 mya to produce darkly pigmented skin in early members of the genus Human being
  10. ^ Bower, C.; Stanley (1992). "The part of nutritional factors in the aetiology of neural tube defects". Journal of Paediatrics and Kid Health. 28 (i): 12–xvi. doi:10.1111/j.1440-1754.1992.tb02610.x. PMID 1554510. S2CID 45104826.
  11. ^ Minns, R.A. (1996). "Folic acid and neural tube defects". Spinal Cord. 34 (8): 460–465. doi:10.1038/sc.1996.79. PMID 8856852.
  12. ^ Copp; et al. (1998). "Embryonic mechanisms underlying the prevenetion of neural tube defects by vitamins". Mental Retardation and Developmental Disabilities Research Reviews. 4 (4): 264–268. doi:ten.1002/(sici)1098-2779(1998)4:4<264::aid-mrdd5>3.0.co;2-chiliad.
  13. ^ Molloy; Mills, J. Fifty.; Kirke, P. North.; Weir, D. Grand.; Scott, J. M.; et al. (1999). "Folate status and neural tube defects". BioFactors. x (2–3): 291–294. doi:x.1002/biof.5520100230. PMID 10609896. S2CID 20128738.
  14. ^ Lucock, M. (2000). "Folic acid: nutritional biochemistry, molecular biology, and role in disease processes". Molecular Genetics and Metabolism. 71 (1–2): 121–138. doi:10.1006/mgme.2000.3027. PMID 11001804.
  15. ^ William; Rasmussen, S. A.; Flores, A; Kirby, R. S.; Edmonds, L. D.; et al. (2005). "Pass up in the prevalence of spina bifida and anencephaly by race/ethnicity:1995–2002". Pediatrics. 116 (3): 580–586. doi:10.1542/peds.2005-0592. PMID 16140696. S2CID 12765407.
  16. ^ Nielsen; et al. (2006). "The importance of the depth distribution of melanin in skin for DNA protection and other photobiological processes". Journal of Photochemistry and Photobiology B: Biology. 82 (3): 194–198. doi:x.1016/j.jphotobiol.2005.11.008. PMID 16388960.
  17. ^ Jane, Higdon. "Vitamin D". Micronutrient Information Center. Linus Pauling Institute. Retrieved ten Dec 2012.
  18. ^ Holick, Michael F. (21 November 2013). "Bioavailability of Vitamin D and Its Metabolites in Black and White Adults". The New England Journal of Medicine. 369 (21): 2047–2048. doi:10.1056/NEJMe1312291. PMID 24256384.
  19. ^ a b DeVita Raeburn, Elizabeth (xx November 2013). "Os Density College in Blacks, Vitamin D Lower". MedPage Today . Retrieved 19 June 2014.
  20. ^ a b Jablonski, N.G.; Chaplin (2000). "The development of human skin coloration". Journal of Man Evolution. 39 (1): 57–106. doi:10.1006/jhev.2000.0403. PMID 10896812. S2CID 38445385.
  21. ^ Harding, R; Healy, Eastward; Ray, A; Ellis, N; Flanagan, N; Todd, C; Dixon, C; Sajantila, A; et al. (2000). "Evidence for Variable Selective Pressures at MC1R". The American Journal of Homo Genetics. 66 (iv): 1351–61. doi:10.1086/302863. PMC1288200. PMID 10733465.
  22. ^ a b c d eastward f g h O'Neil, Dennis. "Skin Color Adaptation". Human Biological Adjustability: Skin Color as an Adaptation. Palomar. Archived from the original on xviii December 2012. Retrieved ten Dec 2012.
  23. ^ O'Neil, Dennis. "Overview". Modern Human being Variation. Palomer. Archived from the original on 5 November 2012. Retrieved 10 Dec 2012.
  24. ^ a b c d e f k h i j k 50 grand Nina, Jablonski (2004). "The development of human being peel and skin color". Annual Review of Anthropology. 33: 585–623. doi:10.1146/annurev.anthro.33.070203.143955. S2CID 53481281.
  25. ^ a b Gina, Kirchweger. "The Biological science of Peel Color: Black and White". Development Library. PBS. Retrieved 10 December 2012.
  26. ^ Jablonski, N.G. (2006). Skin: a Natural History . Berkeley: Academy of California Press.
  27. ^ Dawkins, Richard (2004). The Ancestor's Tale . ISBN9780618619160.
  28. ^ Montagna, W. "The consequences of having naked pare". Nascency Defects: Original Article Series. 17: ane–seven.
  29. ^ Langbein; Rogers, M. A.; Praetzel, S; Cribier, B; Peltre, B; Gassler, N; Schweizer, J; et al. (2005). "Characterization of a novel homo type II epithelial keratin K1b, specifically expressed in eccrine sweat glands". Journal of Investigative Dermatology. 125 (three): 428–444. doi:10.1111/j.0022-202X.2005.23860.x. PMID 16117782.
  30. ^ Blum, H.F. (1961). "Does the melanin pigment of man peel accept adaptive value?". Quarterly Review of Biology. 36: fifty–63. doi:10.1086/403275. PMID 13870200. S2CID 30402725.
  31. ^ a b Rigel, D.South. (2008). "Cutaneous ultraviolet exposure and its relationship to the development of skin cancer". Periodical of the American Academy of Dermatology. 58 (5): S129–S132. doi:10.1016/j.jaad.2007.04.034. PMID 18410798.
  32. ^ a b Jemal; et al. (2001). "Contempo trends in cutaneous melanoma incidence among white in the United States". Journal of the National Cancer Institute. 93 (9): 678–683. doi:10.1093/jnci/93.9.678. PMID 11333289.
  33. ^ Jablonski, Nina. "Department of Anthropology at Penn Country". Penn State University. Archived from the original on 16 January 2013. Retrieved 14 December 2012.
  34. ^ Appenzeller Tim (2012). "Man migrations: Eastern odyssey". Nature. 485 (7396): 24–26. Bibcode:2012Natur.485...24A. doi:10.1038/485024a. PMID 22552074.
  35. ^ a b c d e f chiliad h i j k l Jablonski, Nina (2012). Living Colour. Berkeley, Los Angeles, London: University of California Press. ISBN978-0-520-25153-iii.
  36. ^ "Effects of Ecology and Climate on Human Physical Variations". Retrieved x December 2012.
  37. ^ Miyamura; et al. (2007). "Regulation of human skin pigmentation and responses to ultraviolet radiation". BioFactors. 20 (one): ii–13. doi:ten.1111/j.1600-0749.2006.00358.x. PMID 17250543.
  38. ^ Saraiya; Glanz, Grand; Briss, P. A.; Nichols, P; White, C; Das, D; Smith, S. J.; Tannor, B; Hutchinson, A. B.; Wilson, Thousand. Grand.; Gandhi, N; Lee, Northward. C.; Rimer, B; Coates, R. C.; Kerner, J. F.; Hiatt, R. A.; Buffler, P; Rochester, P; et al. (2004). "Interventions to forestall skin cancer by reducing exposure to ultraviolet radiations: a systematic review". American Journal of Preventive Medicine. 27 (5): 422–466. doi:10.1016/j.amepre.2004.08.009. PMID 15556744.
  39. ^ Agar, N.; Young, A. R. (2005). "Melanogenesis: a photoprotective response to DNA damage?". Mutation Enquiry. 571 (1–2): 121–132. doi:x.1016/j.mrfmmm.2004.11.016. PMID 15748643.
  40. ^ Pfeifer; Yous, Y. H.; Besaratinia, A; et al. (2005). "Mutations induced by ultraviolet light". Mutation Inquiry. 571 (1–two): xix–31. doi:10.1016/j.mrfmmm.2004.06.057. PMID 15748635.
  41. ^ Rouzaud; et al. (2005). "MC1R and the response of melanocytes to ultraviolet radiation". Mutation Research/Primal and Molecular Mechanisms of Mutagenesis. 133–152. 571 (one–two): 133–52. doi:x.1016/j.mrfmmm.2004.09.014. PMID 15748644.
  42. ^ Brenner, M.; Hearing, V. J. (2008). "The protective role of melanin confronting UV impairment in human being skin". Photochemistry and Photobiology. 84 (three): 539–549. doi:10.1111/j.1751-1097.2007.00226.10. PMC2671032. PMID 18435612.
  43. ^ Van Nieuwpoort; Smit, Due north. P.; Kolb, R; Van Der Meulen, H; Koerten, H; Pavel, S; et al. (2004). "Tyrosine-induced melanogenesis shows differences in morphologic and melanogenic preferences of melanosomes from lite and dark pare types". Periodical of Investigative Dermatology. 122 (5): 1251–1255. doi:10.1111/j.0022-202X.2004.22533.x. PMID 15140229.
  44. ^ Kielbassa; Epe, B; et al. (2000). "DNA damaged induced past ultraviolet and visible low-cal and its wavelength dependence". Methods in Enzymology. 319: 436–445. doi:10.1016/s0076-6879(00)19041-10. PMID 10907532.
  45. ^ Cleaver and Crowely (2002). "UV harm, DNA repair and pare carcinogenesis". Frontiers in Bioscience. seven (1–3): 1024–1043. doi:ten.2741/cleaver. PMID 11897551. S2CID 10115842.
  46. ^ Sinha; et al. (2002). "UV-induced DNA damage and repair: a review". Photochemical & Photobiological Sciences. one (four): 225–236. doi:10.1039/b201230h. PMID 12661961. S2CID 11348959.
  47. ^ Schreier, Due west. J.; Schrader, T. E.; Koller, F. O.; Gilch, P; Crespo-Hernández, C. Eastward.; Swaminathan, V. N.; Carell, T; Zinth, W; Kohler, B; et al. (2007). "Thymine dimerization in DNA is an ultrafast photoreaction". Science. 315 (5812): 625–629. Bibcode:2007Sci...315..625S. doi:10.1126/science.1135428. PMC2792699. PMID 17272716.
  48. ^ Epel; et al. (1999). "Development in the floating earth: defenses of eggs and embryos against damage from UV radiations". American Zoologist. 39 (2): 271–278. doi:10.1093/icb/39.ii.271.
  49. ^ Haass, Nikolas K.; Smalley, Keiran S. M.; Li, Ling; Herlyn, Meenhard (1 June 2005). "Adhesion, migration and advice in melanocytes and melanoma". Paint Cell Research. 18 (iii): 150–159. doi:10.1111/j.1600-0749.2005.00235.x. ISSN 0893-5785. PMID 15892711.
  50. ^ Thong, H.Y.; et al. (2003). "The patterns of melanosome distribution in keratinocytes of human skin equally one determining factor of skin colour". British Journal of Dermatology. 149 (3): 498–505. doi:ten.1046/j.1365-2133.2003.05473.10. PMID 14510981. S2CID 43355316.
  51. ^ a b Tadokoro, T; et al. (2005). "Mechanisms of skin tanning in different racial/ethnic groups in response to ultraviolet radiations". Journal of Investigative Dermatology. 124 (vi): 1326–1332. doi:10.1111/j.0022-202X.2005.23760.x. PMID 15955111.
  52. ^ Minwala, Southward; et al. (2001). "Keratinocytes Play a Part in Regulating Distribution Patterns of Recipient Melanosomes in Vitro". Journal of Investigative Dermatology. 117 (two): 341–347. doi:10.1046/j.0022-202x.2001.01411.x. PMID 11511313.
  53. ^ Szabo, G; et al. (1969). "Racial differences in the fate of melanosomes in human being epidermis". Nature. 222 (5198): 1081–1082. Bibcode:1969Natur.222.1081S. doi:10.1038/2221081a0. PMID 5787098. S2CID 4223552.
  54. ^ Lewis, Ricki (2012). Human genetics : concepts and applications (10th ed.). New York, NY: McGraw-Hill Co. pp. 135–136. ISBN978-0-07-352530-3.
  55. ^ "Skin Color" (PDF).
  56. ^ Rana, B. K.; Hewett-Emmett, D.; Jin, L.; Chang, B. H.; Sambuughin, N.; Lin, 1000.; Watkins, Southward.; Bamshad, M.; Jorde, L. B. (1 April 1999). "High polymorphism at the man melanocortin ane receptor locus". Genetics. 151 (4): 1547–1557. doi:10.1093/genetics/151.4.1547. ISSN 0016-6731. PMC1460552. PMID 10101176.
  57. ^ "Furnishings of Ecology and Climate on Human Concrete Variations". world wide web.culturechange.org.
  58. ^ Khan, Razib (2009). "Genetics of human pigmentation: Gene expression". Observe Magazine. Retrieved 11 Dec 2012.
  59. ^ Lamason, R. L.; Mohideen, MA; Mest, JR; Wong, AC; Norton, HL; Aros, MC; Jurynec, MJ; Mao, X; et al. (2005). "SLC24A5, a Putative Cation Exchanger, Affects Pigmentation in Zebrafish and Humans". Science. 310 (5755): 1782–17886. Bibcode:2005Sci...310.1782L. doi:10.1126/scientific discipline.1116238. PMID 16357253. S2CID 2245002.
  60. ^ Gibbons, A. (2007). "AMERICAN ASSOCIATION OF PHYSICAL ANTHROPOLOGISTS Meeting: European Skin Turned Pale Only Recently, Cistron Suggests". Science. 316 (5823): 364. doi:10.1126/scientific discipline.316.5823.364a. PMID 17446367. S2CID 43290419.
  61. ^ "Graphical display of Allele Frequencies for Ala111Thr". Allele Frequency Database. Retrieved 10 October 2012.
  62. ^ "ALFRED – Polymorphism Information – Ala111Thr". Allele Frequency Database. Retrieved 10 Oct 2012.
  63. ^ Pagani L, Kivisild T, Tarekegn A, Ekong R, Plaster C, Romero IG, Ayub Q, Mehdi SQ, Thomas MG, Luiselli D, Bekele E, Bradman N, Balding DJ, Tyler-Smith C (21 June 2012). "Ethiopian Genetic Diversity Reveals Linguistic Stratification and Complex Influences on the Ethiopian Gene pool". The American Periodical of Homo Genetics. 91 (1): 83–96. doi:10.1016/j.ajhg.2012.05.015. PMC3397267. PMID 22726845.
  64. ^ "Dark-skinned immigrant urged to take vitamin D". CBC News. Retrieved 10 December 2012.
  65. ^ Buccimazza SS, Molteno CD, Dunnem TT, Viljoen DL (1994). "Prevalence of neural tube defects in Greatcoat Town, S Africa". Teratology. 50 (3): 194–199. doi:ten.1002/tera.1420500304. PMID 7871483.
  66. ^ "Dark-skinned immigrants urged to take vitamin D". CBC News.
  67. ^ Oglesby, Erika. "Darker Skin? More than Vitamin D, Please!". Care2. Retrieved ane January 2013.
  68. ^ Murray, F. G. (1934). "Pigmentation, sunlight, and nutritional disease". American Anthropologist. 36 (3): 438–445. doi:10.1525/aa.1934.36.iii.02a00100.
  69. ^ Loomis, West. F. (1967). "Skin-pigment regulation of vitamin-D biosynthesis in man". Science. 157 (3788): 501–506. Bibcode:1967Sci...157..501F. doi:x.1126/scientific discipline.157.3788.501. PMID 6028915. S2CID 41681581.
  70. ^ Chaplin Grand, Jablonski NG (2009). "Vitamin D and the evolution of human depigmentation". American Journal of Physical Anthropology. 139 (4): 451–461. doi:10.1002/ajpa.21079. PMID 19425101.
  71. ^ Vieth, R (2003). In Os Loss and Osteoporosis: an Anthropological Perspective. Kluwer Academic/Plenum Press. pp. 135–150.
  72. ^ Garland CF, Garland FC, Gorham ED, et al. (2006). "The Part of Vitamin D in Cancer Prevention". American Periodical of Public Wellness. 96 (2): 252–261. doi:10.2105/ajph.2004.045260. PMC1470481. PMID 16380576.
  73. ^ Armada, J.C. (2008). "Molecular actions of vitamin D contributing to cancer prevention". Molecular Aspects of Medicine. 29 (6): 388–396. doi:10.1016/j.mam.2008.07.003. PMC2613446. PMID 18755215.
  74. ^ Grant, W.B. (2008). "Solar Ultraviolet Irradiance and Cancer Incidence and Mortality". Solar ultraviolet irradiance and cancer incidence and morality. Advances in Experimental Medicine and Biology. Vol. 624. pp. xvi–30. doi:10.1007/978-0-387-77574-6_2. ISBN978-0-387-77573-9. PMID 18348444.
  75. ^ Chen, T.C.; et al. (2007). "Factors that influence the cutaneous synthesis and dietary sources of vitamin D". Archives of Biochemistry and Biophysics. 460 (2): 213–217. doi:10.1016/j.abb.2006.12.017. PMC2698590. PMID 17254541.
  76. ^ Kim, Dae Hyun; Sabour, Siamak; Sagar, Utpal Due north.; Adams, Suzanne; Whellan, David J. (one Dec 2008). "Prevalence of hypovitaminosis D in cardiovascular diseases (from the National Wellness and Nutrition Examination Survey 2001 to 2004)". The American Journal of Cardiology. 102 (11): 1540–1544. doi:x.1016/j.amjcard.2008.06.067. ISSN 1879-1913. PMID 19026311.
  77. ^ McGrath, J.J.; et al. (2004). "Vitamin D – implications for brain development". Periodical of Steroid Biochemistry and Molecular Biology. 89–90 (1–v): 557–560. doi:10.1016/j.jsbmb.2004.03.070. PMID 15225838. S2CID 19993730.
  78. ^ Harms, M.; et al. (2008). "Developmental vitamin D deficiency alters adult behaviour in 129/SvJ and C57BL/6J mice". Behavioural Encephalon Inquiry. 187 (2): 343–350. doi:ten.1016/j.bbr.2007.09.032. PMID 17996959. S2CID 8107153.
  79. ^ "How to get your vitamin D". Archived from the original on xiii January 2013. Retrieved 31 December 2012.
  80. ^ Painter, Kim (xix April 2009). "Your Health". U.s. Today.
  81. ^ "Vitamin D deficiency and skin dominicus exposure". Chicago Tribune. 26 October 2011.
  82. ^ Villarosa, Linda. "Why Black People Demand More Vitamin D". The Root. Archived from the original on 17 November 2012. Retrieved 1 Jan 2013.
  83. ^ "Micronutrient Information Heart". Linus Pauling. Retrieved 1 January 2013.
  84. ^ Marks, Jonathan. "Interview with Jonathan Marks". Race – The Power of an Illusion. PBS. Retrieved 3 January 2013. Certainly dark skin is present all over the world in dissimilar populations. Indigenous Australians, indigenous peoples of India, ethnic peoples of Africa are all very darkly pigmented even though they're not particularly closely related.
  85. ^ "Modernistic man variation: overview". Archived from the original on 18 December 2012.
  86. ^ Chadysiene R, Girgzdys A (2008). "Ultraviolet radiation albedo of natural surfaces". Journal of Environmental Engineering and Landscape Management. 16 (two): 83–88. doi:x.3846/1648-6897.2008.sixteen.83-88.
  87. ^ Krulwich, Robert (2 February 2009). "Your Family May One time Have Been A Dissimilar Colour". NPR . Retrieved 4 July 2013.
  88. ^ "Aboriginal Genome" (PDF).
  89. ^ "Classified Advertising". eight Nov 1828. p. ane – via Trove.
  90. ^ "Aboriginal identity goes beyond skin colour". The Sydney Morning Herald.
  91. ^ "Papua Web" (PDF).
  92. ^ Matisoo-Smith E, Robins JH (2004). "Origins and dispersals of Pacific peoples: Evidence from mtDNA phylogenesis of the pacific rat". Proceedings of the National Academy of Sciences. 101 (24): 9167–9172. doi:10.1073/pnas.0403120101. PMC428491. PMID 15184658.
  93. ^ Norton HL, Friedlaender JS, Merriwether DA, Koki G, Mgone CS, Shriver MD (2006). "Skin and pilus pigmentation variation in island Melanesia". American Periodical of Concrete Anthropology. 130 (2): 254–268. doi:10.1002/ajpa.20343. PMID 16374866.
  94. ^ Sindya N. Bhanoo (3 May 2012). "Another Genetic Quirk of the Solomon Islands: Blond Hair". The New York Times . Retrieved 3 May 2012.
  95. ^ Dupree, L. "Afghānistān: (iv.) ethnography". In Ehsan Yarshater (ed.). Encyclopædia Iranica (Online ed.). U.s.a.: Columbia University. Retrieved five November 2011.
  96. ^ Redd AJ, Stoneking Grand (1999). "Peopling of Sahul: mtDNA Variation in Ancient Australian and Papua New Guinean Populations". American Journal of Human Genetics. 65 (3): 808–828. doi:10.1086/302533. PMC1377989. PMID 10441589.
  97. ^ "Modern man variation: overview". Archived from the original on 5 November 2012.
  98. ^ "blackness Africa | Definition of blackness Africa in English by Lexico Dictionaries". Lexico Dictionaries | English.
  99. ^ Rogers, Alan R.; Iltis, David; Wooding, Stephen (1 February 2004). "Genetic Variation at the MC1R Locus and the Time since Loss of Human Body Pilus". Current Anthropology. 45 (one): 105–108. doi:10.1086/381006. ISSN 0011-3204. S2CID 224795768.
  100. ^ a b Relethford, JH (2000). "Homo skin color multifariousness is highest in sub-Saharan African populations". Homo Biology; an International Record of Research. 72 (5): 773–lxxx. PMID 11126724.
  101. ^ Wilson, James F.; Weale, Michael East.; Smith, Alice C.; Gratrix, Fiona; Fletcher, Benjamin; Thomas, Marker G.; Bradman, Neil; Goldstein, David B. (2001). "Population genetic structure of variable drug response". Nature Genetics. 29 (3): 265–ix. doi:ten.1038/ng761. PMID 11685208. S2CID 25627134. 62% of the Ethiopians fall in the first cluster, which encompasses the majority of the Jews, Norwegians and Armenians, indicating that placement of these individuals in a 'Black' cluster would be an inaccurate reflection of the genetic structure. Only 24% of the Ethiopians are placed in the cluster with the Bantu
  102. ^ Mohamoud, A. M. (October 2006). "P52 Characteristics of HLA Grade I and Form II Antigens of the Somali Population". Transfusion Medicine. 16 (Supplement s1): 47. doi:10.1111/j.1365-3148.2006.00694_52.x. S2CID 70655900.
  103. ^ Michael F. Holick (2010). Vitamin D: Physiology, Molecular Biological science, and Clinical Applications. Springer. p. 531. ISBN978-1-60327-303-9.
  104. ^ Mishra, Anshuman; Nizammuddin, Sheikh; Mallick, Chandana Basu; Singh, Sakshi; Prakash, Satya; Siddiqui, Niyamat Ali; Rai, Niraj; Carlus, S. Justin; Sudhakar, Digumarthi V. S.; Tripathi, Vishnu P.; Möls, Märt (ane March 2017). "Genotype-Phenotype Study of the Heart Gangetic Evidently in India Shows Association of rs2470102 with Skin Pigmentation". Journal of Investigative Dermatology. 137 (three): 670–677. doi:10.1016/j.jid.2016.10.043. ISSN 0022-202X. PMID 27866970.
  105. ^ Iliescu, Florin Mircea; Chaplin, George; Rai, Niraj; Jacobs, Guy South.; Mallick, Chandana Basu; Mishra, Anshuman; Thangaraj, Kumarasamy; Jablonski, Nina G. (2018). "The influences of genes, the environment, and social factors on the development of skin color diversity in India". American Journal of Human Biology. 30 (5): e23170. doi:10.1002/ajhb.23170. hdl:20.500.11820/435c03a5-a3ca-4046-aa50-c81c38d08645. ISSN 1520-6300. PMID 30099804. S2CID 51966049.
  106. ^ Vaughan, Alden T. (1 Oct 1982). "From White Human to Redskin: Changing Anglo-American Perceptions of the American Indian". The American Historical Review. 87 (four): 918. doi:10.2307/1857900. ISSN 0002-8762. JSTOR 1857900.
  107. ^ a b Reference SNP(refSNP) Cluster Written report: rs1426654 **clinically associated** Archived 5 December 2017 at the Wayback Machine.. Ncbi.nlm.nih.gov (30 Dec 2008). Retrieved 2011-02-27.
  108. ^ "Loftier UV occurs throughout the Altiplano region" (PDF).
  109. ^ Thompson, Helen. "A DNA Search for the First Americans Links Amazon Groups to Ethnic Australians". Smithsonian.
  110. ^ "Reconstructing the Deep Population History of Central and Southward America".

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